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Getilapson Ultimate Immune Platinum


  • Purpose: Dietary Supplement
  • Active Ingredients: One Vegetarian capsule, Calcium 87mg, Inositol Hexaphosphate 510mg
  • Directions: For adults: take one vegetarian capsule 2 to 3 times daily,  preferably with a meal. As a reminder, discuss the supplements and medications you take with your health care providers. Store in a dry place and avoid excessive heat.
SKU: GT0309 Category: Tag:


Research & Study

Prostate cancer and inositol ​hexaphosphate: efficacy and mechanisms.

Abstract There are now extensive scientific data suggesting the potential role of dietary and non-dietary phytochemicals in the prevention and control of prostate cancer (PCA) growth and progression. PCA is a disease of elderly male populations with a relatively slower rate of growth and progression as compared to most other cancers and, therefore, is a candidate disease for preventive intervention. Overall, PCA growth and progression involve aberrant mitogenic and survival signaling and deregulated cell cycle progression, accompanied by gradual accumulation of genetic and epigenetic changes over a period of years. Several mechanisms, including overexpression of growth, survival and angiogenic factors and their receptors, together with a loss/decrease of tumor suppressor p53, retinoblastoma and cyclin-dependent kinase inhibitor, have been implicated in PCA growth and progression. Therefore, phytochemicals targeting these molecular events could have a promising role in PCA prevention and/or therapy. Inositol hexaphosphate is a major constituent of most cereals, legumes, nuts, oil seeds and soybean. Taken orally as an over-the-counter dietary/nutrient supplement, and is recognised as offering several health benefits without any known toxicity. In vitro anticancer efficacy of INOSITOL-HEXAPHOSPHATE has been observed in many human, mouse and rat prostate cancer cells. Completed studies also show that oral feeding of INOSITOL-HEXAPHOSPHATE inhibits human PCA xenograft growth in nude mice without toxicity. In a recently completed pilot study, we observed similar preventive effects of INOSITOL-HEXAPHOSPHATE on prostate tumorigenesis in the TRAMP model. Mechanistic studies indicate that INOSITOL-HEXAPHOSPHATE targets mitogenic and survival signaling, as well as cell cycle progression, in PCA cells. INOSITOL-HEXAPHOSPHATE is also shown to target molecular events associated with angiogenesis. Moreover, INOSITOL-HEXAPHOSPHATE has pleiotropic molecular targets for its overall efficacy against PCA and, therefore, could be a suitable candidate agent for preventive intervention of this malignancy in humans. PMID: 16080543[PubMed – indexed for MEDLINE] http://www.ncbi.nlm.nih.gov/pubmed/16080543

Protective effect of inositol hexaphosphate against UVB damage in HaCaT cells and skin carcinogenesis in SKH1 hairless mice.

Williams KA1, Kolappaswamy K, Detolla LJ, Vucenik I. Author information 

Protective effect of inositol hexaphosphate against UVB damage in HaCaT cells and skin carcinogenesis in SKH1 hairless mice. Williams KA1, Kolappaswamy K, Detolla LJ, Vucenik I. Author informationAbstract: UVB radiation damages keratinocytes, potentially inducing chronic skin damage, cutaneous malignancy, and suppression of the immune system. Naturally occurring agents have been considered for prevention and treatment of various kinds of cancer, including skin cancer. INOSITOL-HEXAPHOSPHATE, an antioxidant, is a naturally occurring polyphosphorylated carbohydrate that has shown a strong anticancer activity in several experimental models. We assessed the protective effects of INOSITOL-HEXAPHOSPHATE against UVB irradiationinduced injury and photocarcinogenesis by using HaCaT cells (human immortalized keratinocytes) and SKH1 hairless mice. We found that INOSITOL-HEXAPHOSPHATE counteracts the harmful effects of UVB irradiation and increases the viability and survival of UVB-exposed cells. Treatment with INOSITOL-HEXAPHOSPHATE after UVB irradiation (30 mJ/cm(2)) arrested cells in the G(1) and G(2) M phases while decreasing the S phase of the cell cycle. Treatment with INOSITOL-HEXAPHOSPHATE also decreased UVB-induced apoptosis and caspase 3 activation. Topical application of INOSITOL-HEXAPHOSPHATE followed by exposure to UVB irradiation in SKH1 hairless mice decreased tumor incidence and multiplicity as compared with control mice. Our results suggest that INOSITOL-HEXAPHOSPHATE protects HaCaT cells from UVB-induced apoptosis and mice from UVB-induced tumors. PMID:21819680[PubMed – indexed for MEDLINE] http://www.ncbi.nlm.nih.gov/pubmed/21819680 PMCID: PMC3060432
Antiplatelet activity of inositol hexaphosphate.
Antiplatelet activity of inositol hexaphosphate. Vucenik I1, Podczasy JJ, Shamsuddin AM. Author informationAbstract: Platelet adhesion to endothelial cells, their aggregation and subsequent release of platelet-derived mediators are key steps in the pathogenesis of thrombosis and atherosclerosis. Using impedance technology the effect of INOSITOL-HEXAPHOSPHATE on platelet aggregation and adenosine triphosphate (ATP) release were simultaneously measured in whole blood obtained from healthy volunteers (n = 10). The platelets were activated with adenosine diphosphate (ADP) (10 microM), collagen (2 micrograms/mL), or thrombin (1 U/mL) in the presence or absence of INOSITOL-HEXAPHOSPHATE. INOSITOL-HEXAPHOSPHATE significantly inhibited platelet aggregation induced with all agonists in a dose-response manner (p < 0.0001 for ADP and collagen, p = 0.0103 for thrombin), with the IC50 values of 0.9, 1.6 and 0.8 mM. Secretion of platelet dense granule content was measured in parallel. INOSITOL-HEXAPHOSPHATE strongly and significantly reduced agonist-induced ATP release (p = 0.00247 for ADP; p = 0.0074 for collagen; p = 0.0069 for thrombin). These data demonstrate that INOSITOL-HEXAPHOSPHATE effectively inhibits human platelet aggregation in vitro, suggesting its potential in reducing the risk for cardiovascular disease.PMID: 10625941[PubMed – indexed for MEDLINE] http://www.ncbi.nlm.nih.gov/pubmed/10625941

Additional information

Weight120 oz
Dimensions15 × 15 in

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